Download PDF by G.D. Aurbach (Eds.): Advances in Research and Applications

By G.D. Aurbach (Eds.)

ISBN-10: 0080866395

ISBN-13: 9780080866390

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While the evidence to date implicates an interaction between glucocorticoids and glucose-initiated responses, it is not clear whether this a direct interaction. 5 ? 0 . 6 t 0 . 2 ~ a Values represent means 2 SE for five rats in each group. Food intake was measured over the 3-hour period following intraperitoneal injection of 2-DG (360 mg/kg body weight) of ad libitum-fed rats. Adrenalectomies were performed 7 days prior to the experiment. Data are from J. Allars and D. A. York (unpublished observations).

1986). There is increased sensitivity to norepinephrine and reduced sensitivity to acetylcholine. , 1984). It is possible that these metabolites may be involved in modulating normal feeding patterns through the medial hypothalamus. If so, VMH lesions would be expected to abolish these control signals. This hypothesis requires further testing. c. Hormonal Signals. , 1986). This observation has led to testing the role of hormones that might be released from the gastrointestinal tract as potential signals for modifiers of food intake.

Atkinson and Brent (1982) have found an appetite-suppressing activity in the serum of rats with an intestinal bypass, but its nature is not known. , 1978). Koopmans et al. (1982) have moved 5- and 10-cm segments of ileum to the duodenum (ileal transposition) and found a similar loss of body weight and a decrease in food intake of rats with knife cut-induced hyperphagia and obesity. Again, the simplest explanation for this response to ileal transposition may be the aversive nature of having a shorter small intestine.

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Advances in Research and Applications by G.D. Aurbach (Eds.)


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